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That risk differential increased to 14 percent a year after they stopped a GLP-1, and rose to as much as 22 percent after two years off the medication

GLP-1 receptor agonists reduce inflammation in the brain and the protein-deposit changes characteristic of Alzheimer's disease in animal models, and early-stage clinical trials of older drugs in the same class have produced suggestions of preservation of cognitive function in mild memory and thinking problems and in Parkinson's disease, although large-scale confirmation is still not there [13]

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The most common are mild and temporary