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The failure of proteostasis mechanisms under metabolic stress contributes to the accumulation of misfolded proteins, defective organelles, and disrupted signaling pathways, thereby amplifying metabolic dysfunction and tissue damage (Fig
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received grants to the institution from Amgen, AstraZeneca, Bayer, Boehringer Ingelheim and NovoNordisk and speaker and advisor honoraria from AstraZeneca, Bayer, Boehringer Ingelheim, Eli Lilly, Novo Nordisk, Servier and TAD
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This framework helps providers assess whether dosing adjustments are needed and when to recheck thyroid function
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