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Participants were randomised across placebo, fixed doses of 1, 3, 6 and 9 mg, and two dose-escalation schemes

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These are research comparisons, not proven treatment combinations

Notably, this occurred in the absence of improved insulin sensitivity, neuroinflammation, brain mTORC1 signaling, or activation of brain autophagy all physiological and molecular changes that have been hypothesized to explain the ability of CR to slow the development or promote the clearance of A plaques