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In animal cells the fate of ITCs is well characterized: conjugation to glutathione by GSTs resulting in its intracellular depletion and subsequent degradation of dithiocarbamates through enzymatic events catalysed by -glutamyltranspeptidase, cysteinylglycinase and N -acetyltransferase via the mercapturic acid pathway 76

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These inflammatory cells exacerbate systemic and airway-specific inflammation, disrupting adipose tissue function and impacting other organs [46]

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The Osmolyte TMAO modulates protein folding cooperativity by altering global protein stability