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NAC exerts its pharmacological effects primarily through its reactive thiol (-SH) group, which participates in redox reactions, including the reduction of disulfide bonds and free radical scavenging (15,16)
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Vascular risk factors include stroke, hypertension, hypercholesterolemia, diabetes, and obesity, and amyloid is deposited in the blood vessels of AD patients [102]
The increased PEP also inhibited phosphofructose kinase (encoding by pfk ) activity, which was responsible for the decreased glycolytic flux and the down regulation of the glycolytic activities [33]
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