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ACSL4 exacerbates ischemic stroke by promoting ferroptosis-induced brain injury and neuroinflammation
As was described above, GSR is localized in different subcellular compartments and may differentially regulate NADPH levels
This pathway primarily influences insulin secretion and glucagon inhibition in pancreatic beta and alpha cells, respectively
(April 16, 2025)
NADPH contributes to recycling in oxidized antioxidant systems involving glutathione, thioredoxins, glutaredoxins, and peroxiredoxins and maintains another reactive oxygen species (ROS)-detoxifying enzyme, catalase, in its active form, suggesting that the homeostatic level of G6PD-derived NADPH determines the rate of oxidative damage [8]
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