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Our previous work concluded that this was a consequence of glucagon deficiency rather than improved insulin sensitivity or secretion, as determined by a hyperinsulinaemiceuglycaemic clamp and an IVGTT [9]

Malila, Y., Owolabi, I

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HMGB1 is passively secreted by injured and necrotic cells while it is actively secreted by activated immunocompetent cells such as the dendritic cells and macrophages