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This hypothesis, while speculative, could explain the peptides ability to normalize both deficiency and excess states across diverse neurotransmitter disturbances

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This stability allows GHK-Cu to function as a controlled copper delivery system, silencing coppers potentially harmful redox activity while enabling safe transport into cells where copper serves as a crucial enzyme cofactor

Addressing Deficiencies: Injections are often more effective for severe deficiencies or absorption challenges

Critical Limitations Missing Pharmacokinetic Data: The researchers did not measure actual GHK-Cu concentrations in the brain