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Following cerebral ischemia, the substantial release of various cytokines, chemokines and damage-associated molecular patterns (DAMPs) attributes to the activation and recruitment of neutrophils from the bone marrow, spleen, and peripheral circulation to the site of damage ( Table 2 )
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There have been several cardiovascular outcomes trials (CVOTs) involving GLP-1RAs, which have supported the overall cardiovascular benefits of these drugs
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