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But true dermal repair requires a deeper biological signal
Heart failure (HF) is currently considered a systemic multi-organ syndrome fundamentally driven by metabolic failure, and carnitines, a crucial metabolite in fatty acid metabolism, transport long-chain fatty acids into myocardial mitochondria to produce energy as a source of ATP generation [1] (Fig
A phase II proof-of-concept trial investigated afatinib in pretreated metastatic ESCC patients with confirmed EGFR overexpression
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Ossendorp, F., Mengede, E., Camps, M., Filius, R
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