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Further mutants are obviously necessary to decipher the exact role of Tyr110 and to precisely map the glutathione interaction sites in future studies

References Abdullah, S

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doi:10.1093/humrep/14.7.1690

These results suggest that Sema can counteract the TAC-induced activation of the PI3K/AKT/Creb5/NR4a1 signaling pathway and reduce NR4a1 nuclear-mitochondrial translocation to ameliorate TAC-induced cardiac remodeling