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It is hoped that this review will convince scientists and clinicians to look past paracellular permeability to other mechanisms by which endotoxin is taken into the body in the hope that interventions can be devised that prevent endotoxin uptake and that mechanistic interpretations of processes that increased paracellular permeability can be made
It's important to note that while AOD9604 shows promise, further large-scale human clinical trials are necessary to fully establish its efficacy and long-term safety profile
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Wakabayashi, N., et al., Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation
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