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The diabetic neuropathies
31 Our findings suggest that reduced levels of anorexigenic gut hormones may account, at least in part, for this observation
Nitric oxide generated by activated inducible Nitric oxide synthase reacts with specific cysteine residues (e.g., Cys166) of ERO1 which leads to the S-nitrosylation of ERO1 and reduces the production of oxidants in the ER, thus causing ER reducing stress and finally promoting the aging process of cells ( ERO1 may be a potential therapeutic target in hereditary myopathies ( 2+ dynamics between ER and mitochondria, and subsequently affecting muscle cell bioenergetics and muscle contractile function
PeruzzoloTLPintoJVRozaTHShintaniAOAnzolinAPGnielkaVet al
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