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Primary mechanisms include: Because of this profile, AOD 9604 is commonly used in in vitro systems and animal models focused on obesity research, adipocyte signaling, metabolic dysfunction, and energy-balance regulation

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Baseline measurements should confirm AMPK suppression (reduced phosphorylation status in muscle or liver) and NAD+ depletion (low tissue NAD+ levels or elevated NNMT enzymatic activity)

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AOD 9604 had no effect on serum IGF-1 levels at any dose tested