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The pathological mechanism of HD involves both a loss of function, due to impaired activity of the normal HTT protein, and a toxic gain of function from the mutant protein [91], resulting in oxidative stress, synaptic and mitochondrial dysfunction, impaired nuclear pore complex function, altered axonal trafficking, transcriptional dysregulation, and dysfunction of the ubiquitinproteasome pathway [92]
Chiarla C et al (2010) Severe hypocholesterolemia in surgical patients, sepsis, and critical illness
Intramuscular (IM) Injection This is the most common method
Improvements in known cardiometabolic risk factors amongst adults with type 2 diabetes may have a beneficial effect on downstream disease burden [3]
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