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This clinical study highlights how low GSH is associated with multiple age-related diseases and shows that GSH supplementation in type 2 diabetes reduced oxidative DNA damage and may help support glycemic control
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Cell culture studies demonstrate stimulation of beta-3 adrenergic receptors in adipocytes, enhancing fat oxidation while inhibiting lipogenesis via acetyl CoA carboxylase blockade
The net result is a shift toward fat oxidation rather than fat storage
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