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6) yielded outputs X(GD_Sh2)a and X(GD_Sh2)b that retained activity on GD_SS1.1, GD_SS1.2 and GD_SS2, indicating that further selection was necessary (Extended Data Fig
Also, in the experiments on human breast cancer cells, Won and Seo found that the inactivation of PI3K/Akt/mTORC1 signaling pathway promotes the expression of caspases and Bax leading to increased apoptosis [302]
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