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In particular, a growing body of in vivo evidence supports the concept that some cancers become functionally dependent on FSP1, creating a therapeutic vulnerability that can be exploited either as monotherapy in specific contexts or as combination therapy with ferroptosis-inducing treatments

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Picture this: as fat cells expand, they trigger an overproduction of NNMT, slowing down fat cell metabolism the very essence of fat burning

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Without timely treatment, NAFLD can progress to cirrhosis, hepatocellular carcinoma, and even death (Friedman et al., 2018)