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When administered therapeutically, GLP-1 receptor agonists work through several mechanisms: Enhancing insulin secretion from pancreatic beta cells in a glucose-dependent manner [1] [4] Suppressing glucagon release from alpha cells, reducing hepatic glucose output Slowing gastric emptying , which helps moderate post-meal blood glucose rises Promoting satiety through central nervous system pathways, potentially supporting weight reduction Investigational transdermal GLP-1 delivery systems typically utilise specialised technologies such as microneedles or active delivery methods rather than conventional patches, as GLP-1 molecules are large peptides that do not easily penetrate the skin barrier
Lower temperatures slow molecular motion, which in turn reduces the rate of degradation reactions
Additionally, DSIP has been linked to regulation of oxidative stress pathways through upregulation of antioxidant enzyme systems such as superoxide dismutase and glutathione peroxidase [4]
Nat Rev Neurol (2015) 11(12):71124
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