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The cancer connection is currently theoretical but plausible given these biological effects

MOTS-c modulates the FOXO transcription factors and regulates cellular senescence, the state in which aged or damaged cells stop dividing but refuse to die, instead secreting a cocktail of inflammatory signals known as the senescence-associated secretory phenotype (SASP)

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Under experimentally induced oxidative stress conditions using hydrogen peroxide, BPC 157 appeared to support fibroblast survival relative to untreated controls