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Mechanisms of plaque vulnerability and rupture

The accumulation of oxidized protein products may lead to changes in their tertiary structure, leading to protein dysfunction (impairing metabolic functions in yeast cells) [14]

Acetyl-l-carnitineamelioratesmitochondrial damage and apoptosis following spinal cord injury in rats

NPM1 mutations are known to result in aberrant cytoplasmic localization and downregulation of the NPM1 protein 28 , as observed in our data (log 2 fold change (FC): 0.44, P 0.2 for C3, C7 and C8)

described a case of a 40-year-old test subject who experienced priapism as a result of self-administering melanotan I [20]