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L., & Garca, M

141 They remain in the body, interacting with GLP-1 receptors in various organs, which could lead to DDIs through mechanisms of action such as slowing gastric emptying, reducing fat mass and inflammation, and increasing glomerular filtration rate (GFR) and renal plasma flow, consequently altering the pharmacokinetics of the victim drug (Figure 3)

(2023) "Pharmacotherapy of obesity: an update on the available medications and drugs under investigation" eClinicalMedicine, 58, 101882 5

At the WT GIP receptor, both endogenous agonists GIP(1-42) and GIP(1-30)NH 2 , and tirzepatide stimulated a concentration-dependent increase in signaling for all pathways measures (Figures 1AE)

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