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Figure 9 summarizes the key pathological mechanisms linking EC metabolic dysregulation to atherosclerosis and highlights three categories of therapeutic interventionstraditional drugs, emerging metabolic targets, and microbiota-derived therapiescurrently under investigation

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They remain in a state of readiness, a kind of alert hibernation, and their energy consumption remains low

This process upregulates GLS expression and promotes the proliferation, migration, and Gln metabolism of gastric cancer (GC) cells