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Antidotal therapy, a clinical approach against toxicity, is widely inaccessible and also unreliable, and current approaches to chemically modifying acetaminophen, what my research seeks to do, are impractical since they sacrifice acetaminophens pain-relieving properties. The problem was clear, but the solution wasnt so simple
GIPR monoagonism drives weight loss and food intake reduction on its own in both humans (52) and rodents (5355)
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