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Yang HN, Choi JH, Park JS, Jeon SY, Park KD, Park KH
10.1111/jog.12050 156
Retrieved 9 April 2022
256 Conclusion and Future Directions TMAO may drive the cross-talk between inflammatory response and oxidative stress by mediating cell death (including apoptosis, pyroptosis, autophagy, and ferroptosis, etc) and then induce pathological processes such as foam cell activation, massive secretion of cytokines and adhesion molecules, overaccumulation of ROS, enhanced platelet reactivity and abnormal vascular tone regulation
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