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These processes commonly result in increased bioavailability of ROS due to excess ROS formation, decreased nitric oxide (NO) levels, and impaired antioxidant capacity in the arteries, heart, brain, and kidneys (133, 134)

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Furthermore, in superoxide dismutase 1-induced neurodegenerative model rats, neurons in the developed amyotrophic lateral sclerosis were found to express increased LOX expression levels and exhibit increased enzyme activity (54)

retinoic acid receptor activation), so they complement each other rather than compete