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1994 Aug 1;301 ( Pt 3)(Pt 3):769-75
Frontiers in pharmacology , 11 , 579944
As part of its wound healing effect, GHK-Cu has been shown to contribute to the initial upregulation of growth factors and inflammatory mediators during nerve regeneration [8]
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Similar patterns of severe mitochondrial impairment have been extensively documented in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS)
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