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This increases IGF1R transcription, mediates the PI3K/Akt and Ras/ERK signaling pathways, and enhances the proliferation, differentiation, and anti-apoptotic abilities of HCC cells both in vitro and in vivo, ultimately promoting HCC progression [131]
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disposition model, which were effects of body weight on clearance (CL), inter-compartment CL ( Q ), central volume ( V c ) and peripheral volume ( V p ), and effect of glycaemic status on CL
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