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277,278 Complementing these cellintrinsic models, Farrell extended network adaptation to the tumor microenvironment, arguing that resistance is ecologically driven by crosstalk among cancer cells, CAFs, and macrophages via paracrine signals and exosomes, enabling noncellautonomous drug tolerance
IL-4 plays a crucial role in establishing an immunosuppressive TME by promoting macrophage polarization toward an M2-like phenotype, which contributes to ICB resistance in OvCa
Together, these results indicate that GSH supports lipid abundance by preventing NRF2 activation in the liver
GSH is essential for maintaining a healthy immune system
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