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Mutant P53 leads to defects in growth arrest, senescence, and apoptosis but can sensitize cells to ferroptosis [9]
In a postweaning androgenization (PWA) mouse model, treatment with a GLP-1/GIP/glucagon triagonist significantly reduced ovarian weight and serum luteinizing hormone (LH) levels
Oxidative stress can be increased by persistent inflammation brought on by autoimmune illnesses
Preserved frontal memorial processing for pictures in patients with mild cognitive impairment
[U- 13 C]glutamine isotope tracing experiments showed that GGCT overexpression reduced Gln contribution to the TCA cycle and diverted it preferentially to the GSH synthesis pathway for ROS regulation
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