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What is the regulatory status of GHK-Cu
The ferroptosis of TECs is also regulated by the interactive influence of the immune microenvironment (201)
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however, in acute inflammation models, excessive release of HMGB1 can activate caspase-1 through the AIM2 inflammasome, promote the maturation and release of IL-1 and IL-18, and exacerbate tissue damage (190)
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