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Since glucocorticoids are known to mediate inhibition of NF-kB (beneficially modulated by certain antioxidants such as thiols), adjunctive therapy with an effective thiol donor (e.g., NAC or SAMe) and vitamin E may optimize control of inflammatory mediator release and cell response as compared to pure immunomodulatory treatment or therapy limited to antioxidants

GHK-Cu is non-toxic in vitro at nanomolar to micromolar concentrations

105,106,326 Targeting key signaling pathways, such as TGF-, Wnt, and Notch, and EMT-related transcription factors (e.g., ZEB1, TWIST1) is another strategy under active investigation