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Discussion A few cases of FSH-secreting gonadotroph cell adenoma manifesting as ovarian hyperstimulation have been reported with the endocrinological characteristics, but the true incidence of this condition including unnoticed or subclinical cases remains unclear [1, 711]
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Side-by-side comparisons over several months reveal changes that day-to-day observation misses
As an example, GSH depletion has been shown to promote pro-inflammatory microglial phenotypes and amplify astrocyte-microglia crosstalk (Vilhardt et al., 2017)
However, permeability increases measured in most studies are secondary to epithelial damage, which allows non-selective flux via the unrestricted pathway
Biologic and pharmacologic regulation of mammalian glutathione synthesis
