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It is composed of glutamic acid, cysteine and glycine and exists in almost every cell of the body
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Tumors with HRD rely on alternative DNA repair mechanisms for survival, so agents that inhibit DNA single-strand break (SSB) repair or induce more SSBs trigger genomic instability and apoptosis 34
The possible mechanism might be the interference of those drugs with sodium retention and cardiac inflammation, microvascular sparseness and fibrosis [123]
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