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10.1186/s12943-020-01258-7 Summary Keywords ulcerative colitis, GHK-Cu, SIRT1/STAT3 pathway, co-culture system, network pharmacology, molecular docking Citation Mao S, Huang J, Li J, Sun F, Zhang Q, Cheng Q, Zeng W, Lei D, Wang S and Yao J (2025) Exploring the beneficial effects of GHK-Cu on an experimental model of colitis and the underlying mechanisms
Patients not only improved their blood sugar levels but also lost meaningful weight
The thiol group of C32 in PfGrx therefore facilitates the C29-dependent deglutathionylation of roGFP2(SSG) without forming an intramolecular disulfide bond
It supports cellular energy production (ATP) to improve cell turnover
In the current study, we clearly demonstrated that NAFLD induced by long-term HFD consumption was associated with increased hepatic protein-SSG formation
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