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C., Nemutlu, E., & Atilla, P

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Dampened ER stress signaling was also evident from diminishment of the splicing of the IRE1 nuclease target Xbp1 (Figure 1B) and of the upregulation of the stress-regulated factor CHOP (Figure 1C)

Once activated, these caspases cleave gasdermins to liberate the pore-forming N-terminal domains, enabling their targeting of cellular membranes to form large transmembrane pores, which disrupts cellular electrochemical gradient, and leads to water influx and cell swelling (160, 161)