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10.1016/j.yrtph.2020.104665 105 YoussefA

Moreover, increased gene expression of MLCK pointed toward an activation of the leak pathway through contraction of the cytoskeleton

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Chiechio S, Copani A, De Petris L, et al

[1] At the cellular level, the tendon work is genuinely mechanistic: in cultured rat tendon fibroblasts, BPC-157 accelerated cell outgrowth, improved survival under oxidative stress, and increased migration in a dose-dependent way, apparently through the FAKpaxillin pathway