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Jul 2022;130(7):76001
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Animal studies have demonstrated that short-chain fatty acid (SCFA) levels decrease during the early phase after stroke, and stroke-induced gut dysbiosis is associated with metabolic alterations linked to enhanced inflammatory signaling (179, 180)
SIRT1 deacetylates SREBP-1c and ChREBP, suppressing the expression of their downstream lipogenic genes, ultimately inhibiting lipogenesis and mitigating MAFLD [28,29]
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