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J Clin Med [Internet]
Although GIP receptor activation may exert anti-emetic effects through brainstem modulation, these effects appear insufficient to completely counterbalance GLP-1 mediated emesis during early treatment and dose escalation ( In contrast, glucagon receptor activation is independently associated with nausea and vomiting through both central and peripheral mechanisms, including effects on gastric motility and autonomic signaling
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This process also helps reduce inflammation , which contributes to less post-treatment discomfort
Sometimes, patients must undergo tests and assessments to determine the appropriate medication and dosage
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