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Exposing cerebral mitochondria to THC was reported to induce a ten-fold increase in free radical leak and increased H 2 O 2 production [40], while in epithelial cells, THC upregulated inflammation-related genes and downregulated antioxidant-related genes [34]
Astrocytes maintain their antioxidative functions through two primary mechanisms: the Nrf2keap1antioxidant response element (ARE) pathway and glutathione (GSH) synthesis
Antioxidant and Anti-Fibrotic Effects: GHK-Cu provides copper-dependent activation of superoxide dismutase (SOD) and other antioxidant enzymes that neutralize reactive oxygen species
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Ayton S, Lei P, Duce JA, Wong BXW, Sedjahtera A, Adlard PA et al
