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May help support glutathione conjugation in the clearing toxins
These results suggest that upon depletion of GSH, tissues undergo reprogramming to hyperactivate NRF2, possibly as a mechanism to upregulate adjacent antioxidant pathways and buffer oxidative stress induced upon GSH depletion
In conclusion, our study provides a robust methodology for redox profiling and establishes baseline data for NAD and glutathione metabolites in healthy populations
Its application to deeper knee structures such as the joint capsule, ligaments, or cartilage in human patients remains largely theoretical at this stage
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