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Overexpression of the PI3K/Akt signaling mechanism in breast cancer results in chemotherapeutic resistance, as it interacts with other pathways such as MAPK and Wnt/-catenin [86,87]
Presented for research literature review only Wellner VP et al
[1] Selectivity testing indicated the scaffold did not inhibit related SAM-dependent methyltransferases or NAD+ salvage enzymes
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