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Furthermore, metabolic pathways associated with IL1A, IL1B, and IL1RN were predominantly enriched in TCMR cells, suggesting that the intensity of inflammation in TCMR is linked to amino sugar and nucleotide metabolism ( Figures 5GI )
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(1996) first demonstrated that TNF- induces insulin resistance by promoting serine phosphorylation of insulin receptor substrate 1(IRS-1), thereby inhibiting the tyrosine kinase activity of the insulin receptor (44)
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