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The loss of DJ-1-mediated maintenance of the transsulfuration pathway could render microglia more vulnerable to ferroptotic damage, thereby linking its classical cytoprotective role in redox homeostasis directly to the emerging pathway of ferroptosis in neurodegeneration
Simultaneously, the resulting increase in IGF-1 supports the preservation and hypertrophy of lean muscle tissue
Glutathione contents of tissues in the aging mouse
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