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indeed, an SLC1A5-deficient mouse model shows decreased induction of T helper 1 (Th1) and Th17 cells but not Th2, regulatory T (Treg), or CD8(+) T cells, leading to a decrease in proinflammatory T-cell responses 134

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Thank I'm Andrew Huberman, and I'm a professor of neurobiology and ophthalmology at Stanford School of Medicine

Gene-expression research suggests GHK-Cu influences the activity of more than 60 genes involved in tissue remodeling, antioxidant defense, and wound repair, effectively nudging cells toward a regenerative rather than degenerative program