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The mechanism proposed involves chromatin remodeling at the hTERT promoter specifically, a reduction in the repressive histone modifications (H3K9me3, H3K27me3) that maintain transcriptional silencing in somatic cells allowing activating transcription factors to access promoter binding sites and initiate hTERT transcription
also showed that miR-146a levels were upregulated in the vitreous humor (VH) and serum of UM patients, which could have a regulatory role in UM melanocyte surveillance (75)
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