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Mears MJ, Bandaru P, Neczypor EW, Byun MK, Zhang M, Gorr MW, et al
High homocysteine concentrations may promote the development of atherosclerosis (atherogenesis) and thrombogenesis via mechanisms involving oxidative stress and endothelial dysfunction, inflammation, abnormal blood coagulation, and disordered lipid metabolism (reviewed in 35)
AMPK suppresses cholesterol and TG synthesis by inhibiting HMGCR [11]
Dysregulation of phosphorylation-dependent signaling has been implicated in numerous human diseases, including cancer, diabetes, autoimmune disorders, and neurodegenerative diseases, making protein phosphorylation a central focus of modern chemical biology, biochemistry, and drug discovery
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