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Gut bacteria from multiple sclerosis patients modulate human T cells and exacerbate symptoms in mouse models
References Goodman & Gilmans The Pharmacological Basis of Therapeutics Katzung Basic and Clinical Pharmacology Harrisons Principles of Internal Medicine WHO Model Formulary and Drug Monographs
(14) Assisting with regular cell death
No GHK-Cus mechanism targets tissue remodeling through fibroblast activation and matrix metalloproteinase regulation, which requires localized tissue concentration, not systemic plasma levels
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