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Neuroprotection and Cognitive Function DSIP has shown some potential as a neuroprotective agent, particularly in protecting the brain from damage caused by ischemia or neurodegeneration

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These are plausible risk signals, not confirmed IGF-1 LR3-specific findings

It is known that excess lipid accumulation in non-adipose tissues is associated with higher concentrations of NEFA and TAG in the circulation, and that this is often accompanied by inflammation, oxidative stress and cellular dysfunction or death ( Reference Mattern and Hardin 53 Reference Perez-Martinez, Perez-Jimenez and Lopez-Miranda 55 )