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Each has its pros and cons, but they are not created equal in terms of convenience or effectiveness

GLP-1 exerts its effects through several key mechanisms: Glucose-dependent insulin secretion : GLP-1 stimulates pancreatic beta cells to release insulin, but only when blood glucose levels are elevated, reducing the risk of hypoglycaemia Glucagon suppression : It inhibits the release of glucagon from pancreatic alpha cells, thereby reducing hepatic glucose production Delayed gastric emptying : GLP-1 slows the rate at which food leaves the stomach, leading to more gradual glucose absorption and increased satiety Appetite regulation : Acting on brain centres, GLP-1 promotes feelings of fullness and reduces food intake Prescription GLP-1 receptor agonists (such as semaglutide, dulaglutide, and liraglutide) are synthetic analogues designed to mimic and enhance these natural effects

The Role of Dietitians in GLP-1 Nutrition, Side-Effect Management and Food Relationships Many people stop GLP1 medications early because of side effects, unrealistic expectations or feeling unsure how to eat on them

All of the velocities are given by Michaelis-Menten kinetics, with the K m values taken from the literature, and where available, from human data

K., Valentine, J